Jeff Childers explains ….the best explanation…so even a “Scientist” can understand

Remember how Moderna and Pfizer always said that their mRNA platforms were so great because they could just quickly reprogram them to mint new variant-specific versions of their vaccines whenever we needed them? And how the president of Pfizer said in December that they’d have an Omicron-specific version out as soon as January or February?

And then … nothing happened?

I’ve wondered several times on the blog about where those promised variant-specific vaccines were hiding. Well, a new study seems to provide some answers. The bottom line is, the new versions are here but they DON’T WORK.

The study, quietly published earlier this year, bears the somewhat opaque title, “mRNA-1273 or mRNA-Omicron boost in vaccinated macaques elicits comparable B cell expansion, neutralizing antibodies and protection against Omicron.” It weighs in at a blockbuster 66 authors, including NIAID and NIH scientists.

The researchers compared the rates of waning antibodies in monkeys after being vaccinated with the original mRNA jab to the rate of waning antibodies after vaccination with the variant-specific version. They were surprised to find that there was no additional benefit from the new vaccine version over the original, even though the monkeys in both groups were infected with Omicron and not the original Wuhan strain.

They concluded, “Significant and equivalent control of virus replication in lower airways was observed following either boost.”

So that wasn’t good for business, and explained why we haven’t seen any variant-specific versions. But then things got even more interesting. The 66 authors invoked OAS, original antigenic sin, as a LIKLEY potential explanation for why the newer drugs don’t work against the new variants. Likely.

The observation that boosting with either mRNA-1273 or mRNA-Omicron resulted in the expansion of a similarly high frequency of cross-reactive B cells likely stems from the principle of original antigenic sin, otherwise termed antigenic imprinting, whereby prior immune memory is recalled by a related antigenic encounter.

For new readers, original antigenic sin is a problem observed in some leaky vaccines. What happens is, a person’s immune system gets locked into its original response and never learns any new responses when it encounters variants. In other words, because of the leaky vaccine (the “original sin”), the person’s immune system only makes antibodies to the ORIGINAL viral strain, and never updates its antibodies at all to match new variant types, like an unvaccinated immune system can and will easily do.

In other words, injectees’ immune systems are no longer trainable against the disease.

Because the locked immune systems spur genetic development of the target virus, over time the original antibodies become increasingly less effective against new variant types. This can leave the vaccinated people in a really difficult spot. In bad cases, it basically makes them into AIDS patients as to the particular bug they got vaccinated against. In other words, they become immunocompromised to that particular disease, doomed to keep catching it over and over, with each case getting more and more severe.

Since the OAS issue was not the authors’ primary research objective, they didn’t chase it down to prove whether OAS caused the failure of the variant-specific jabs or not. They could have, but they didn’t. For some reason.

But they did say, “likely.